Rabbit Polyclonal Antibody to RT4I1 (C-term)
货号:
P30847
别名:
Reticulon-4-interacting protein 1, mitochondrial, NOGO-interacting mitochondrial protein, RTN4IP1, NIMP
应用:
WB,IHC,FCM
反应种属:
Human, Mouse
抗体类型:
Primary antibody
Swissprot:
Q8WWV3
规格:
目录价
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Description |
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This gene encodes a novel mitochondrial protein that interacts with reticulon 4, which is a potent inhibitor of regeneration following spinal cord injury. The interaction of reticulon 4 with mitochondrial proteins may provide insight into the mechanisms for reticulon-induced inhibition of neurite growth. |
Specification |
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Aliases | Reticulon-4-interacting protein 1, mitochondrial, NOGO-interacting mitochondrial protein, RTN4IP1, NIMP |
Entrez GeneID | 84816 |
Swissprot | Q8WWV3 |
WB Predicted band size | 43.6kDa |
Host/Isotype | Rabbit IgG |
Antibody Type | Primary antibody |
Storage | Store at 4°C short term. Aliquot and store at -20°C long term. Avoid freeze/thaw cycles. |
Species Reactivity | Human, Mouse |
Immunogen | This RT4I1 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 335-363 amino acids from the C-terminal region of human RT4I1. |
Formulation | Purified antibody in PBS with 0.05% sodium azide. |
Application |
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WB | 1/1000 |
IHC | 1/100-1/500 |
FCM | 1/10-1/50 |
Product Image
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RT4I1 Antibody (C-term) (Cat. #P30847) western blot analysis in WiDr cell line lysates (35ug/lane).This demonstrates the RT4I1 antibody detected the RT4I1 protein (arrow).
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RT4I1 Antibody (C-term) (Cat. #P30847) flow cytometric analysis of WiDr cells (right histogram) compared to a negative control cell (left histogram).FITC-conjugated goat-anti-rabbit secondary antibodies were used for the analysis.
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RT4I1 Antibody (C-term) (Cat. #P30847) immunohistochemistry analysis in formalin fixed and paraffin embedded human hepatocarcinoma followed by peroxidase conjugation of the secondary antibody and DAB staining. This data demonstrates the use of the RT4I1 Antibody (C-term) for immunohistochemistry. Clinical relevance has not been evaluated.